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- 3-Hydroxy-3′, 4′-Dimethoxyflavone Suppresses Bcl-W-Induced Invasive Potentials And Stemness In Glioblastoma Multiforme
- 44 Over-Expression Of Microrna-122 (Mir-122) Induces Apoptosis By Targeting Cycling1 And Bcl-W Genes In Vitro
- A Novel Bcl-2/Bcl-Xl/Bcl-W Inhibitor Abt-737 As Therapy In Multiple Myeloma
- A Retrograde Neuronal Survival Response: Target-Derived Neurotrophins Regulate Mef2D And Bcl-W
- Abstract A3: Bcl-W-Induced Invasive Pathways In Gastric Cancer Cells
- Adeno-Associated Virus-Mediated Delivery Of Bcl-W Gene Improves Outcome After Transient Focal Cerebral Ischemia
- Akt Regulates Drug-Induced Cell Death Through Bcl-W Downregulation
- Anti-Apoptotic Functions Of Bcl-W, Mcl-1, And A1 In Mitotic Cell Death
- Apoptosis Regulator Bcl-W Is Essential For Spermatogenesis But Appears Otherwise Redundant
- At‐101 Downregulates Bcl 2 And Mcl 1 And Potentiates The Cytotoxic Effects Of Lenalidomide And Dexamethasone In Preclinical Models Of Multiple Myeloma And W …
- Bcl-2 Family Compensation Regulates T Cell Homeostasis And Reveals A Bim: Bcl-W Axis In T-All
- Bcl-2 Is A Better Abt-737 Target Than Bcl-Xl Or Bcl-W And Only Noxa Overcomes Resistance Mediated By Mcl-1, Bfl-1, Or Bcl-B
- Bcl-W (Edding) With Mitochondria
- Bcl-W Buries Its Tail, Lets Cell Die
- Bcl-W Deficiency Perturbs Enterocyte Apoptosis And Proliferation
- Bcl-W Enhances Mesenchymal Changes And Invasiveness Of Glioblastoma Cells By Inducing Nuclear Accumulation Of Β-Catenin
- Bcl-W Expression And Localization In Brain Ischemia
- Bcl-W Expression In Colorectal Adenocarcinoma
- Bcl-W Expression Is Increased In Brain Regions Affected By Focal Cerebral Ischemia In The Rat
- Bcl-W Forms Complexes With Bax And Bak, And Elevated Ratios Of Bax/Bcl-W And Bak/Bcl-W Correspond To Spermatogonial And Spermatocyte Apoptosis In The Testis
- Bcl-W Has A Fundamental Role In B Cell Survival And Lymphomagenesis
- Bcl-W Is A Regulator Of Microtubule Inhibitor-Induced Mitotic Cell Death
- Bcl-W Is An Important Determinant Of Damage-Induced Apoptosis In Epithelia Of Small And Large Intestine
- Bcl-W Is Expressed In A Majority Of Infiltrative Gastric Adenocarcinomas And Suppresses The Cancer Cell Death By Blocking Stress-Activated Protein Kinase/C-Jun Nh2 …
- Bcl-W Promotes Cell Invasion By Blocking The Invasion-Suppressing Action Of Bax
- Bcl-W Promotes Cell Invasion By Inducing Mitochondrial Ros Production
- Bcl-W Promotes Gastric Cancer Cell Invasion By Inducing
- Bcl-W Promotes Gastric Cancer Cell Invasion By Inducing Matrix Metalloproteinase-2 Expression Via Phosphoinositide 3-Kinase, Akt, And Sp1
- Bcl-W Promotes Gastric Cancer Cell Invasion By Inducing Matrix Metalloprotenase-2 Expression Via Phosphoinositide 3-Kinase, Akt, And Sp1
- Bcl-W Protects Hippocampus During Experimental Status Epilepticus
- Bcl-W Significantly Contributes To B-Cell Lymphoma Survival And Development
- Bcl-W, A Novel Member Of The Bcl-2 Family, Promotes Cell Survival.
- Bcl-W, A Radio-Resistant Protein, Promotes The Gastric Cancer Cell Migration By Inducing The Phosphorylation Of Focal Adhesion Kinase
- Cloning And Characterization Of The 5′ Utr Of The Rat Anti-Apoptotic Bcl-W Gene
- Co-Crystallization With Conformation-Specific Designed Ankyrin Repeat Proteins Explains The Conformational Flexibility Of Bcl-W
- Co-Inhibition Of Bcl-W And Bcl2 Restores Antiestrogen Sensitivity Through Becn1 And Promotes An Autophagy-Associated Necrosis
- Crystal Structure Of A Bcl-W Domain-Swapped Dimer: Implications For The Function Of Bcl-2 Family Proteins
- Design Of Druglike Small Molecules For Possible Inhibition Of Antiapoptotic Bcl-2, Bcl-W, And Bfl-1 Proteins
- Detergent N-Octyl-Β-D-Maltoside (Om) Induces Both Monomerization And Oligomerization Of Bcl-W
- Developmental Regulation Of The Bcl-2 Family During Spermatogenesis: Insights Into The Sterility Of Bcl-W−/− Male Mice
- Differential Expression Of Bcl-W And Bcl-X Messenger Rna In The Developing And Adult Rat Nervous System
- Directed Elimination Of Senescent Cells By Inhibition Of Bcl-W And Bcl-Xl
- Dynamic Changes Of The Anti‐And Pro‐Apoptotic Proteins Bcl‐W, Bcl‐2, And Bax With Smac/Diablo Mitochondrial Release After Photothrombotic Ring Stroke In Rats
- Effect Of Iodide On Fas, Fas-Ligand And Bcl-W Mrna Expression In Thyroid Of Nod Mice Pretreated With Methimazole
- Effect Of Massive Small Bowel Resection On The Bax/Bcl-W Ratio And Enterocyte Apoptosis
- Epidermal Growth Factor Alters The Bax: Bcl-W Ratio Following Massive Small Bowel Resection
- Epidermal Growth Factor Receptor Signaling Regulates Bax And Bcl-W Expression And Apoptotic Responses During Intestinal Adaptation In Mice
- Estrogen Regulates Bcl-W And Bim Expression: Role In Protection Against Β-Amyloid Peptide-Induced Neuronal Death
- Expression Of Bcl-W In Gastric Carcinoma And Associated Clinicopathologic Factors
- Expression Of Bcl-W Protein In Human Small Intestinal Adenocarcinoma And Effect Of Bcl-W Sirna On Apoptosis In Intestinal Adenocarcinoma Hutu-80 Cells
- Expression Of Concern: Akt Regulates Drug-Induced Cell Death Through Bcl-W Downregulation
- Functional Characterization Of Two Splice Variants Of Rat Bad And Their Interaction With Bcl-W In Sympathetic Neurons
- Functional Investigation Of The Bcl-W Promoter During Neuronal Differentiation And Survival
- Hypermethylation Of Mir-205-5P By Ir Governs Aggressiveness And Metastasis Via Regulating Bcl-W And Src
- Increased Bcl-W Expression Following Focally Evoked Limbic Seizures In The Rat
- Inhibition Of Neurotensin Receptor 1 Induces Intrinsic Apoptosis Via Let-7A-3P/Bcl-W Axis In Glioblastoma
- Lncrna Rp11-436H11. 5, Functioning As A Competitive Endogenous Rna, Upregulates Bcl-W Expression By Sponging Mir-335-5P And Promotes …
- Long Noncoding Rna Hotair Modulates Mir-206-Mediated Bcl-W Signaling To Facilitate Cell Proliferation In Breast Cancer
- Met/Hgf Receptor Modulates Bcl-W Expression And Inhibits Apoptosis In Human Colorectal Cancers
- Microrna Alteration After H_2O_2-Induced Pc12 Cells Apoptosis And Regulatory Mechanism Of Bcl-W
- Microrna-125B Promotes Apoptosis By Regulating The Expression Of Mcl-1, Bcl-W And Il-6R
- Microrna-203 Is A Prognostic Indicator In Bladder Cancer And Enhances Chemosensitivity To Cisplatin Via Apoptosis By Targeting Bcl-W And Survivin
- Microrna‐203 Suppresses Bladder Cancer Development By Repressing Bcl‐W Expression
- Microrna-335 Acts As A Metastasis Suppressor In Gastric Cancer By Targeting Bcl-W And Specificity Protein 1
- Mir-122 Targets An Anti-Apoptotic Gene, Bcl-W, In Human Hepatocellular Carcinoma Cell Lines
- Mir-133B Regulates Bladder Cancer Cell Proliferation And Apoptosis By Targeting Bcl-W And Akt1
- Mir-335 Inhibits The Proliferation And Invasion Of Clear Cell Renal Cell Carcinoma Cells Through Direct Suppression Of Bcl-W
- Mir-335 Represents An Invasion Suppressor Gene In Ovarian Cancer By Targeting Bcl-W
- Mir-340-5P Suppresses Aggressiveness In Glioblastoma Multiforme By Targeting Bcl-W And Sox2
- Mir-497 Induces Apoptosis Of Breast Cancer Cells By Targeting Bcl-W
- Mir-93-5P Suppresses Cellular Senescence By Directly Targeting Bcl-W And P21
- Mirna-107 Enhances Chemosensitivity To Paclitaxel By Targeting Antiapoptotic Factor Bcl-W In Non Small Cell Lung Cancer
- Mirna-195 Sensitizes Human Hepatocellular Carcinoma Cells To 5-Fu By Targeting Bcl-W
- Mitochondrial Fragmentation And Neuronal Cell Death In Response To The Bcl-2/Bcl-Xl/Bcl-W Antagonist Abt-737
- Mitochondrial Morphogenesis, Dendrite Development, And Synapse Formation In Cerebellum Require Both Bcl-W And The Glutamate Receptor Δ2
- Molecular Docking Studies Of Bioactive Compounds From Annona Muricata Linn As Potential Inhibitors For Bcl-2, Bcl-W And Mcl-1 Antiapoptotic Proteins
- Neuroprotective Properties Of Bcl‐W In Alzheimer Disease
- Nf-Κb Activation Fails To Protect Cells To Tnfα-Induced Apoptosis In The Absence Of Bcl-Xl, But Not Mcl-1, Bcl-2 Or Bcl-W
- Nf-Κb P65/P52 Plays A Role In Gdnf Up-Regulating Bcl-2 And Bcl-W Expression In 6-Ohda-Induced Apoptosis Of Mn9D Cell
- Of Apoptosis (Tweak)-Fibroblast Growth Factor-Inducible 14 (Fn14) Signaling System Regulates Glioma Cell Survival Via Nfκb Pathway Activation And Bcl-Xl/Bcl-W …
- Of The Viability Of Cultured Rat Neurons By The Non-Essential Amino Acids L-Serine And Glycine That Upregulates Expression Of The Anti-Apoptotic Gene Product Bcl-W
- Oral Presentations Op01: Neurodegenerative Diseases. Bcl‐W Plays A Key Neuroprotective Role In Alzheimer’S Disease
- Overexpression Of Bcl-W In The Testis Disrupts Spermatogenesis: Revelation Of A Role Of Bcl-W In Male Germ Cell Cycle Control
- Overexpression Of The Cell Death Suppressor Bcl-W In Ischemic Brain: Implications For A Neuroprotective Role Via The Mitochondrial Pathway
- Oxidative Modification Of Mir-184 Enables It To Target Bcl-Xl And Bcl-W
- Pathways In Cutaneous Squamous Cell Carcinoma Cells By Diclofenac/Hyaluronic Acid Is Related To Upregulation Of Bad As Well As Downregulation Of Mcl‐1 And Bcl‐W
- Pharmacological Blockade Of Bcl-2, Bcl-Xl And Bcl-W By The Bh3 Mimetic Abt-737 Has Only Minor Impact On Tumour Development In P53-Deficient Mice
- Preclinical Evaluation Of Drug Combinations Identifies Co-Inhibition Of Bcl-2/Xl/W And Mdm2 As A Potential Therapy In Uveal Melanoma
- Pro-And Anti-Apoptogenic Genes In Response To Benzene Exposure: Immunohistochemical Localization Of P53, Bag, Bad, Bax, Bcl-2, And Bcl-W In Lung Epithelia
- Proapoptotic Bh3-Only Proteins Trigger Membrane Integration Of Prosurvival Bcl-W And Neutralize Its Activity
- Purified Rat Anti-Bcl-W
- Reciprocal Developmental Changes In The Roles Of Bcl-W And Bcl-X (L) In Regulating Sensory Neuron Survival
- Regulation Of Apoptosis By Mir-122 In Pterygium Via Targeting Bcl-W
- Rna Expression Bcl-W, A New Related Protein Bcl-2 Family, And Caspase-3 In Isolated Sertoli Cells From Pre-Pubertal Rat Testes
- Sensory Neuropathy Attributable To Loss Of Bcl-W
- Signaling Components Involved In Bcl-W-Induced Migration Of Gastric Cancer Cells
- Solution Structure Of Human Bcl-W Modulation Of Ligand Binding By The C-Terminal Helix
- Specificity Protein 1 Expression Contributes To Bcl-W-Induced Aggressiveness In Glioblastoma Multiforme
- Src And Epidermal Growth Factor Receptor Mediate The Pro-Invasive Activity Of Bcl-W
- Structural Model Of The Bcl-W−Bid Peptide Complex And Its Interactions With Phospholipid Micelles,
- Surface Plasmon Resonance Study Of Apoptotic Regulators Bcl-2, Bcl-W, Bcl-Xl, Mcl-1 And A1 And The Small Molecule Inhibitor (Smi) Tw-37: The Idea Of A Temporal …
- Survival Activity Of Bcl-2 Homologs Bcl-W And A1 Only Partially Correlates With Their Ability To Bind Pro-Apoptotic Family Members
- The Anti-Apoptotic Molecules Bcl-Xl And Bcl-W Target Protein Phosphatase 1> To Bad
- The Anti‐Apoptotic Molecules Bcl‐Xl And Bcl‐W Target Protein Phosphatase 1Α To Bad
- The Bcl-2/Bcl-Xl/Bcl-W Inhibitor, Navitoclax, Enhances The Activity Of Chemotherapeutic Agents In Vitro And In Vivo
- The Bcl-W Promoter Is Activated By Β-Catenin/Tcf4 In Human Colorectal Carcinoma Cells
- The Effects Of Rapamycin On The Symptoms Of Cerebral Ischemia Due To Changing The Expression Of Mir-1 And Its Target Genes, Bad And Bcl-W
- The Mrna And Protein Expression Of Bcl-W And Its Clinical Value In Breast Cancer
- The Relationship Between The Expression Of Mir-422A In Peripheral Blood And Bcl-W In Cerebral Cortex And Exercise-Induced Central Fatigue [J]
- The Structure Of Bcl‐W Reveals A Role For The C‐Terminal Residues In Modulating Biological Activity
- Tissue Expression And Subcellular Localization Of The Pro-Survival Molecule Bcl-W
- Tweak-Fn14 Ligand-Receptor System Induces Invasive Glioma Cell Survival Via Upregulation Of Bcl-Xl And Bcl-W Protein
- Β-Amyloid-Induced Neuronal Apoptosis Involves C-Jun N-Terminal Kinase-Dependent Downregulation Of Bcl-W