HYPOCRETIN (OREXIN ) deficiency in human narcolepsy HYPOCRETIN (OREXIN ) loss in Parkinson’s disease Neurons containing HYPOCRETIN (OREXIN ) project to multiple neuronal systems Behavioral correlates of activity in identified HYPOCRETIN /OREXIN neurons HYPOCRETIN (OREXIN ) loss in Alzheimer’s disease The HYPOCRETIN /OREXIN system HYPOCRETIN (OREXIN ) cell loss in Parkinson’s disease Release of HYPOCRETIN […]
BRAIN metabolism during FASTING Caloric restriction and intermittent FASTING: two potential diets for successful BRAIN aging FASTING is neuroprotective following traumatic BRAIN injury The Effects of Intermittent FASTING on BRAIN and Cognitive Function FASTING biases BRAIN reward systems towards high‐calorie foods Chronic intermittent FASTING improves cognitive functions and BRAIN structures in mice The FASTING polypeptide
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The HPA axis is involved in the neurobiology of mood disorders and functional illnesses, including anxiety disorder, bipolar disorder, insomnia, posttraumatic stress disorder, borderline personality disorder, ADHD, major depressive disorder, burnout, chronic fatigue syndrome, fibromyalgia, irritable bowel syndrome, and alcoholism.[15] Antidepressants, which are routinely prescribed for many of these illnesses, serve to regulate HPA axis
DYNORPHIN 1–8 and DYNORPHIN 1–9 are ligands for the κ-subtype of opiate receptor Behavioral effects and in vivo degradation of intraventricularly administered DYNORPHIN-(1-13) and D-Ala2-DYNORPHIN-(1-11) in rats DYNORPHIN is a specific endogenous ligand of the kappa opioid receptor Specific receptor for the opioid peptide DYNORPHIN: structure–activity relationships DYNORPHIN-(1-13), an extraordinarily potent opioid peptide Enhancement of
ANTI‐GLYCATIVE and ANTI‐inflammatory effects of caffeic acid and ellagic acid in kidney of diabetic mice ANTI-oxidative, ANTI–GLYCATIVE and ANTI-apoptotic effects of oleanolic acid in brain of mice treated by D-galactose ANTI–GLYCATIVE effects of oleanolic acid and ursolic acid in kidney of diabetic mice ANTI–GLYCATIVE potential of triterpenes: a mini-review ANTI–GLYCATIVE and ANTI-inflammatory effects of protocatechuic
EXTRACELLULAR GLYCATION crosslinks: prospects for removal Attenuation of EXTRACELLULAR MATRIX accumulation in diabetic nephropathy by the advanced GLYCATION end product cross-link BREAKER ALT-711 via a protein kinase C-α … Advanced GLYCATION endproduct CROSSLINKING in the cardiovascular system Advanced GLYCATION End Product Cross‐Linking: Pathophysiologic Role and Therapeutic Target in Cardiovascular Disease GLYCATION end-product cross-link BREAKER reduces
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PROTEIN CARBONYLATION PROTEIN CARBONYLATION in human diseases PROTEIN CARBONYLATION, cellular dysfunction, and disease progression Cell signaling by PROTEIN CARBONYLATION and deCARBONYLATION PROTEIN CARBONYLATION as a major hallmark of oxidative damage: update of analytical strategies Quantification of PROTEIN CARBONYLATION PROTEIN CARBONYLATION and metabolic control systems Rules governing selective PROTEIN CARBONYLATION PROTEIN CARBONYLATION: avoiding pitfalls in the
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CATALASE The mechanism of CATALASE action. I. Steady-state analysis CATALASE activity Some variables affecting the measurement of “CATALASE activity” in soil A spectrophotometric method for measuring the breakdown of hydrogen peroxide by CATALASE Perborate as substrate in a new assay of CATALASE On the mechanism of the decomposition of hydrogen peroxide by CATALASE CATALASE photoinactivation
TIME –Restricted feeding is a preventative and therapeutic intervention against diverse nutritional challenges TIME –Restricted feeding without reducing caloric intake prevents metabolic diseases in mice fed a high-fat diet TIME –Restricted feeding and risk of metabolic disease: a review of human and animal studies TIME –Restricted feeding attenuates age-related cardiac decline in Drosophila TIME –Restricted
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Stress, CORTICOTROPIN –RELEASING FACTOR , and behavior Chemical and biological characterization of CORTICOTROPIN RELEASING FACTOR Psychiatric implications of basic and clinical studies with CORTICOTROPIN –RELEASING FACTOR CORTICOTROPIN –RELEASING FACTOR has an anxiogenic action in the social interaction test Effects of CORTICOTROPIN –RELEASING FACTOR and growth hormone-RELEASING FACTOR on sleep and activity in rats Influence of
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