SCIENCE

NEUROPLASTICITY

DISEASE & DISORDERS, SCIENCE / By

Harnessing NEUROPLASTICITY for clinical applications NEUROPLASTICITY in children Conceptualizing functional NEUROPLASTICITY Homer2 is necessary for EtOH-induced NEUROPLASTICITY Stress, depression, and NEUROPLASTICITY: a convergence of mechanisms Threoretical aspects of NEUROPLASTICITY Toward a theory of NEUROPLASTICITY Antidepressants and NEUROPLASTICITY Mitochondria and NEUROPLASTICITY Central NEUROPLASTICITY and pathological pain NEUROPLASTICITY subserving motor skill learning NEUROPLASTICITY mediated by altered gene […]

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TRYPTOPHAN HYDROXYLASE 2

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TRYPTOPHAN HYDROXYLASE –2 controls brain serotonin synthesis TRYPTOPHAN HYDROXYLASE 2 (TPH2 ) gene variants associated with ADHD TRYPTOPHAN HYDROXYLASE –2 (TPH2 ) in disorders of cognitive control and emotion regulation: a perspective Loss-of-function mutation in TRYPTOPHAN HYDROXYLASE –2 identified in unipolar major depression Amygdala responsiveness is modulated by TRYPTOPHAN HYDROXYLASE –2 gene variation TRYPTOPHAN HYDROXYLASE

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GABA (γ-AMINOBUTYRIC ACID)

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Free γ –AMINOBUTYRIC ACID in brain The action of γ –AMINOBUTYRIC ACID on cortical neurones γ –AMINOBUTYRIC ACID in brain: its formation from glutamic ACID Biochemical-physiology correlations in studies of the γ –AMINOBUTYRIC ACID system Factor I and γ –AMINOBUTYRIC ACID Epilepsy and γ –AMINOBUTYRIC ACID -mediated inhibition The Nature of γ –AMINOBUTYRIC ACID γ

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HPA AXIS

SCIENCE, SOLUTIONS / By

The HPA axis is involved in the neurobiology of mood disorders and functional illnesses, including anxiety disorder, bipolar disorder, insomnia, posttraumatic stress disorder, borderline personality disorder, ADHD, major depressive disorder, burnout, chronic fatigue syndrome, fibromyalgia, irritable bowel syndrome, and alcoholism.[15] Antidepressants, which are routinely prescribed for many of these illnesses, serve to regulate HPA axis

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GLYCATION

DISEASE & DISORDERS, SCIENCE / By

Glycation and diabetic complications Role of glycation in aging Mechanisms of glycation in atherogenesis The pathological implications of protein glycation. Glycation products and the pathogenesis of diabetic complications Free radical generation by early glycation products: a mechanism for accelerated atherogenesis in diabetes Advanced glycation end-products and atherosclerosis Inhibition of protein glycation and advanced glycation end

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TIME RESTRICTED FEEDING

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TIME –Restricted feeding is a preventative and therapeutic intervention against diverse nutritional challenges TIME –Restricted feeding without reducing caloric intake prevents metabolic diseases in mice fed a high-fat diet TIME –Restricted feeding and risk of metabolic disease: a review of human and animal studies TIME –Restricted feeding attenuates age-related cardiac decline in Drosophila TIME –Restricted

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CORTICOTROPIN RELEASING FACTOR

SCIENCE, SOLUTIONS / By

Stress, CORTICOTROPIN –RELEASING FACTOR , and behavior Chemical and biological characterization of CORTICOTROPIN RELEASING FACTOR Psychiatric implications of basic and clinical studies with CORTICOTROPIN –RELEASING FACTOR CORTICOTROPIN –RELEASING FACTOR has an anxiogenic action in the social interaction test Effects of CORTICOTROPIN –RELEASING FACTOR and growth hormone-RELEASING FACTOR on sleep and activity in rats Influence of

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CATALASE

SCIENCE, SOLUTIONS / By

CATALASE The mechanism of CATALASE action. I. Steady-state analysis CATALASE activity Some variables affecting the measurement of “CATALASE activity” in soil A spectrophotometric method for measuring the breakdown of hydrogen peroxide by CATALASE Perborate as substrate in a new assay of CATALASE On the mechanism of the decomposition of hydrogen peroxide by CATALASE CATALASE photoinactivation

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ADIPOCYTE DIFFERENTIATION

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Understanding Adipocyte differentiation Adipocyte differentiation from the inside out Control of Adipocyte differentiation Adipocyte differentiation and gene expression Molecular regulation of Adipocyte differentiation E2Fs regulate Adipocyte differentiation The roles of PPARs in Adipocyte differentiation Failure of Adipocyte differentiation causes type II diabetes mellitus? The role of MAPKs in Adipocyte differentiation and obesity Transcriptional regulation of

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METABOLIC REPROGRAMMING

SCIENCE, SOLUTIONS / By

Metabolic reprogramming: the emerging concept and associated therapeutic strategies HIF1α and Metabolic reprogramming in inflammation Lipid Metabolic reprogramming in cancer cells Metabolic reprogramming: a cancer hallmark even warburg did not anticipate Metabolic reprogramming of the tumor miRNAs link Metabolic reprogramming to oncogenesis Metabolic reprogramming of immune cells in cancer progression The biology of cancer: Metabolic

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